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(hämtat ur Lunds universitets publikationsdatabas)
- Dystrophin deficiency reduces atherosclerotic plaque development in ApoE-null mice.
- Expression of fibromodulin in carotid atherosclerotic plaques is associated with diabetes and cerebrovascular events.
- IL-22 affects smooth muscle cell phenotype and plaque formation in apolipoprotein E knockout mice.
- Regulation of Smooth Muscle Dystrophin and Synaptopodin 2 Expression by Actin Polymerization and Vascular Injury.
- The small leucine-rich repeat proteoglycans in tissue repair and atherosclerosis.
- Fibromodulin Deficiency Reduces Low-Density Lipoprotein Accumulation in Atherosclerotic Plaques in Apolipoprotein E-Null Mice.
- Increased Neointimal Thickening in Dystrophin-Deficient mdx Mice.
- Mobilization of regulatory T cells in response to carotid injury does not influence subsequent neointima formation.
- A constitutive endogenous osteopontin production is important for macrophage function and differentiation.
- Inhibition of injury-induced arterial remodelling and carotid atherosclerosis by recombinant human antibodies against aldehyde-modified apoB-100.
- Involvement of the CD1d-Natural killer T cell pathway in neointima formation after vascular injury
- Fibulin-2 is present in murine vascular lesions and is important for smooth muscle cell migration.
- Tissue fibrocytes in patients with mild asthma: A possible link to thickness of reticular basement membrane?
- Upregulated TRPC1 Channel in Vascular Injury In Vivo and Its Role in Human Neointimal Hyperplasia.