E-post: gabriela [dot] godaly [at] med [dot] lu [dot] se
(hämtat ur Lunds universitets publikationsdatabas)
- A mycobacterial phosphoribosyltransferase promotes bacillary survival by inhibiting oxidative stress and autophagy pathways in macrophages and zebrafish.
- Impaired CXCR1-dependent oxidative defence in active tuberculosis patients.
- Innate immunity and genetic determinants of urinary tract infection susceptibility.
- Simplified citrate anticoagulation for CRRT without calcium replacement.
- Interleukin-8 receptor deficiency confers susceptibility to acute pyelonephritis
- Neutrophil recruitment, chemokine receptors, and resistance to mucosal infection
- The 'innate' host response protects and damages the infected urinary tract
- Toll-like receptor signaling and chemokine receptor expression influence the severity of urinary tract infection
- Innate defences and resistance to gram negative mucosal infection
- Interleukin 8 receptor deficiency confers susceptibility to acute experimental pyelonephritis and may have a human counterpart
- Interleukin-8 receptor knockout mice have subepithelial neutrophil entrapment and renal scarring following acute pyelonephritis
- Mechanisms of Escherichia coli induced transepithelial neutrophil migration
- Transepithelial neutrophil migration is CXCR1 dependent in vitro and is defective in IL-8 receptor knockout mice