046-173 23 3
Majlis [dot] Svensson [at] med [dot] lu [dot] se
Publikationer (hämtat ur Lunds universitets publikationsdatabas)
- Glycolipid receptor depletion as an approach to specific antimicrobial therapy.
- Lipopolysaccharide from enterohemorrhagic Escherichia coli binds to platelets via TLR4 and CD62 and is detected on circulating platelets in patients with hemolytic uremic syndrome.
- Uropathogenic Escherichia coli as a model of host-parasite interaction.
- Expression of nitric oxide synthase isoforms in the mouse kidney: cellular localization and influence by lipopolysaccharide and Toll-like receptor 4.
- Natural history of renal scarring in susceptible mIL-8Rh-/- mice.
- Neutrophil activation and renal scarring - Reply
- Urinary tract infections and the mucosal Immune system
- Carbohydrate receptor depletion as an antimicrobial strategy for prevention of urinary tract infection
- Escherichia coli P fimbriae utilize the Toll-like receptor 4 pathway for cell activation
- Escherichia coli-induced inducible nitric oxide synthase and cyclooxygenase expression in the mouse bladder and kidney
- Fimbriae, transmembrane signaling, and cell activation
- The 'innate' host response protects and damages the infected urinary tract
- pap genotype and P fimbrial expression in Escherichia coli causing bacteremic and nonbacteremic febrile urinary tract infection