Kristian Riesbeck

författare

• 2013
  • Candida albicans is a crafty microbe deceiving its host by using complement regulators and proteases.
  • Haemophilus influenzae acquires vitronectin via the ubiquitous Protein F to subvert host innate immunity.
  • Impact of sequence diversity in the Moraxella catarrhalis UspA2/UspA2H head domain on vitronectin binding and antigenic variation.
  • Outer membrane vesicles shield Moraxella catarrhalis β-lactamase from neutralization by serum IgG.
  • Streptococcus pneumoniae endopeptidase O (PepO): a multifunctional plasminogen and fibronectin binding protein, facilitating evasion of innate immunity and invasion of host cells.
  • The unique structure of Haemophilus influenzae protein E reveals multiple binding sites for host factors.
• 2012
  • A Metalloproteinase Karilysin Present in the Majority of Tannerella forsythia Isolates Inhibits All Pathways of the Complement System.
  • A Role for TLRs in Moraxella-superantigen induced polyclonal B cell activation.
  • Acquisition of Complement Inhibitor Serine Protease Factor I and Its Cofactors C4b-Binding Protein and Factor H by Prevotella intermedia.
  • Comparative Analysis of the Humoral Immune Response to Moraxella catarrhalis and Streptococcus pneumoniae Surface Antigens in Children Suffering from Recurrent Acute Otitis Media and Chronic Otitis Media with Effusion
  • Crystallization and X-ray diffraction analysis of a novel surface-adhesin protein: protein E from Haemophilus influenzae.
  • Enolase of Streptococcus pneumoniae Binds Human Complement Inhibitor C4b-Binding Protein and Contributes to Complement Evasion.
  • Genome Sequence of Moraxella catarrhalis RH4, an Isolate of Seroresistant Lineage
  • Haemophilus influenzae Protein F Mediates Binding to Laminin and Human Pulmonary Epithelial Cells.
  • Haemophilus influenzae Uses the Surface Protein E To Acquire Human Plasminogen and To Evade Innate Immunity
  • Human pathogens utilize host extracellular matrix proteins laminin and collagen for adhesion and invasion of the host.
  • Increase of beta-Lactam-Resistant Invasive Haemophilus influenzae in Sweden, 1997 to 2010
  • Increase of ss-lactam resistant invasive Haemophilus influenzae in Sweden 1997-2010.
  • Moraxella catarrhalis: from interactions with the host immune system to vaccine development.
  • QseC controls biofilm formation of non-typeable Haemophilus influenzae in addition to an AI-2-dependent mechanism.
  • The Activation Pattern of Blood Leukocytes in Head and Neck Squamous Cell Carcinoma Is Correlated to Survival
• 2011
  • A Descriptive Study of Bacterial Load of Full-Thickness Surgical Wounds in Dermatologic Surgery.
  • Bacterial outer membrane vesicles in disease and preventive medicine.
  • Comparative Study of Immune Status to Infectious Agents in Elderly Patients with Multiple Myeloma, Waldenstrom's Macroglobulinemia, and Monoclonal Gammopathy of Undetermined Significance
  • Effects of NOD-like receptors in human B lymphocytes and crosstalk between NOD1/NOD2 and Toll-like receptors.
  • Haemophilus influenzae Protein E Binds to the Extracellular Matrix by Concurrently Interacting With Laminin and Vitronectin
  • Haemophilus influenzae protein E recognizes the C-terminal domain of vitronectin and modulates the membrane attack complex.
  • Immune Evasion of Moraxella catarrhalis Involves Ubiquitous Surface Protein A-Dependent C3d Binding.
  • Invasive disease caused by Haemophilus influenzae in Sweden 1997-2009; evidence of increasing incidence and clinical burden of non-type b strains
  • Moraxella catarrhalis outer membrane vesicles carry beta-lactamase and promote survival of Streptococcus pneumoniae and Haemophilus influenzae by inactivating amoxicillin.
  • Multicomponent Moraxella catarrhalis outer membrane vesicles induce an inflammatory response and are internalized by human epithelial cells.
  • Necrotizing myositis and septic shock caused by Haemophilus influenzae type f in a previously healthy man diagnosed with an IgG3 and a mannose-binding lectin deficiency
  • Nucleotide-binding and oligomerization domain-like receptors and retinoic acid inducible gene-like receptors in human tonsillar T lymphocytes.
  • Temporal development of the humoral immune response to surface antigens of Moraxella catarrhalis in young infants
  • Vitronectin in host pathogen interactions and antimicrobial therapeutic applications
• 2010
  • B cell activation by outer membrane vesicles--a novel virulence mechanism.
  • Binding of complement regulators to invasive nontypeable Haemophilus influenzae is not increased compared to nasopharyngeal isolates, but serum resistance is linked to disease severity.
  • Genome analysis of Moraxella catarrhalis strain RH4, a Human Respiratory Tract Pathogen.
  • Haemophilus influenzae and the complement system.
  • Invasive disease by Haemophilus influenzae in Sweden 1997-2009; evidence of increasing incidence and clinical burden of non-type b strains.
  • Proteases of periodontal pathogens Porphyromonas gingivalis, Tannerella forsythia and Prevotella intermedia target all pathways of the complement system
  • Protein E of Haemophilus influenzae Is a Ubiquitous Highly Conserved Adhesin.
  • The role of complement inhibitor C4b-binding protein in pneumococcal infection
  • Vitronectin binds to the head region of Moraxella catarrhalis ubiquitous surface protein A2 and confers complement-inhibitory activity.
  • Vitronectin in bacterial pathogenesis: A host protein used in complement escape and cellular invasion.
• 2009
  • Clinical isolates of Streptococcus pneumoniae bind the complement inhibitor C4b-binding protein in a PspC allele-dependent fashion.
  • Complement evasion strategies of pathogens-Acquisition of inhibitors and beyond.
  • Expression and function of Nod-like receptors (NLRs) in human lymphocytes
  • Immunoglobulin D enhances immune surveillance by activating antimicrobial, proinflammatory and B cell-stimulating programs in basophils
  • Interpain A, a Cysteine Proteinase from Prevotella intermedia, Inhibits Complement by Degrading Complement Factor C3.
  • Nontypeable Haemophilus influenzae Adhesin Protein E: Characterization and Biological Activity.
  • Nontypeable Haemophilus influenzae Protein E Binds Vitronectin and Is Important for Serum Resistance
  • Superantigen- and TLR-dependent activation of tonsillar B cells after receptor-mediated endocytosis.
  • Toll-like receptor agonists induce inflammation and cell death in a model of head and neck squamous cell carcinomas
  • Virulence mechanisms of Moraxella in the pathogenesis of infection.
• 2008
  • Binding of complement inhibitor C4b-binding protein contributes to serum resistance of Porphyromonas gingivalis.
  • CEACAM1 inhibits Toll-like receptor 2-triggered antibacterial responses of human pulmonary epithelial cells
  • Haemophilus influenzae interacts with the human complement inhibitor factor H
  • Identification of a novel Haemophilus influenzae protein important for adhesion to epithelial cells.
  • Moraxella catarrhalis-dependent tonsillar B cell activation does not lead to apoptosis but to vigorous proliferation resulting in nonspecific IgM production.
  • Moraxella-Dependent {alpha}1-Antichymotrypsin Neutralization - A Unique Virulence Mechanism.
  • Protein D of Haemophilus influenzae: a protective nontypeable H. influenzae antigen and a carrier for pneumococcal conjugate vaccines.
  • Purification of alpha1-antichymotrypsin from human plasma with recombinant M. catarrhalis ubiquitous surface protein A1.
  • S100A7 - a novel protein in allergic rhinitis
  • The Moraxella IgD-binding protein MID/Hag is an oligomeric autotransporter.
• 2007
  • Biphasic Effect of Gingipains from Porphyromonas gingivalis on the Human Complement System.
  • Characterization of the IgD binding site of encapsulated Haemophilus influenzae serotype b.
  • Comparison of an automated Borrelia indirect chemiluminescent immunoassay (CLIA) with a VlsE/C6 ELISA and Immunoblot.
  • Current progress of adhesins as vaccine candidates for Moraxella catarrhalis
  • Current progress of adhesins as vaccine candidates for Moraxella catarrhalis.
  • Haemophilus influenzae Survival during Complement-Mediated Attacks Is Promoted by Moraxella catarrhalis Outer Membrane Vesicles.
  • Interaction with C4b-binding protein contributes to nontypeable Haemophilus influenzae serum resistance.
  • The UspA1 protein of Moraxella catarrhalis induces CEACAM-1-dependent apoptosis in alveolar epithelial cells.
• 2006
  • Alpha1-antitrypsin inhibits Moraxella catarrhalis MID protein-induced tonsillar B cell proliferation and IL-6 release.
  • Haemophilus influenzae Surface Fibrils Contribute to Serum Resistance by Interacting with Vitronectin.
  • Immunomodulation by fluoroquinolones and other antibacterial agents.
  • MID and UspA1/A2 of the human respiratory pathogen Moraxella catarrhalis, and interactions with the human host as basis for vaccine development.
  • Purification of IgD from human serum - A novel application of recombinant M. catarrhalis IgD-binding protein (MID).
  • Serological Response to M. catarrhalis Outer Membrane Protein MID as Compared to UspA1 and A2.
  • Streptococcal M protein: a multipotent and powerful inducer of inflammation.
  • Structure and Immunological Action of the Human Pathogen Moraxella catarrhalis IgD-Binding Protein.
  • The IgD C(H)1 region contains the binding site for the human respiratory pathogen Moraxella catarrhalis IgD-binding protein MID.
  • The IgD-binding domain of the Moraxella IgD-binding protein MID (MID962-1200) activates human B cells in the presence of T cell cytokines.
  • The Respiratory Pathogen Moraxella catarrhalis Binds to Laminin via Ubiquitous Surface Proteins A1 and A2.
  • Wnt-5a mRNA translation is suppressed by the Elav-like protein HuR in human breast epithelial cells.
  • alpha1-Antitrypsin inhibits Moraxella catarrhalis MID protein-induced tonsillar B cell proliferation and IL-6 release.
• 2005
  • A novel localization of the G-protein-coupled CysLT1 receptor in the nucleus of colorectal adenocarcinoma cells.
  • Ionic binding of C3 to the human pathogen Moraxella catarrhalis is a unique mechanism for combating innate immunity
  • The CD19 molecule is crucial for MID-dependent activation of tonsillar B cells from children.
  • The Respiratory Pathogen Moraxella catarrhalis Adheres to Epithelial Cells by Interacting with Fibronectin through Ubiquitous Surface Proteins A1 and A2.
• 2004
  • Dicloxacillin and erythromycin at high concentrations increase ICAM-1 expression by endothelial cells: a possible factor in the pathogenesis of infusion phlebitis.
  • Immunization with the Truncated Adhesin Moraxella catarrhalis Immunoglobulin D-Binding Protein (MID764-913) Is Protective against M. catarrhalis in a Mouse Model of Pulmonary Clearance.
  • Septicemia with Staphylococcus aureus, beta-hemolytic streptococei group B and G, and Escherichia coli in a patient with rheumatoid arthritis treated with a recombinant human interleukin 1 receptor antagonist (Anakinra)
  • The emerging pathogen Moraxella catarrhalis interacts with complement inhibitor C4b binding protein through ubiquitous surface proteins A1 and A2
• 2003
  • Non-typeable Haemophilus influenzae activates human eosinophils through {beta}-glucan receptors.
  • Nontypeable Haemophilus influenzae activates human eosinophils through beta-glucan receptors
  • Paronychia due to Prevotella bivia that resulted in amputation: fast and correct bacteriological diagnosis is crucial.
  • Several gene programs are induced in ciprofloxacin-treated human lymphocytes as revealed by microarray analysis.
  • The Immunoglobulin D-Binding Protein MID from Moraxella catarrhalis Is Also an Adhesin.
  • The Moraxella catarrhalis immunoglobulin D-binding protein MID has conserved sequences and is regulated by a mechanism corresponding to phase variation.
• 2002
  • A pilot with pain in his leg: thigh abscess caused by Salmonella enterica serotype Brandenburg.
  • CTLA4Ig combined with anti-LFA-1 prolongs cardiac allograft survival indefinitely.
  • Comparison of the antibacterial activities of ampicillin, ciprofloxacin, clarithromycin, telithromycin and quinupristin/dalfopristin against intracellular non-typeable Haemophilus influenzae.
  • Immunomodulating activity of quinolones: review.
  • Susceptibilities of Mycobacterium marinum to Gatifloxacin, Gemifloxacin, Levofloxacin, Linezolid, Moxifloxacin, Telithromycin, and Quinupristin-Dalfopristin (Synercid) Compared to Its Susceptibilities to Reference Macrolides and Quinolones.
  • The immunoglobulin D-binding part of the outer membrane protein MID from Moraxella catarrhalis comprises 238 amino acids and a tetrameric structure.
  • The novel IgD binding protein from Moraxella catarrhalis induces human B lymphocyte activation and Ig secretion in the presence of Th2 cytokines.
• 2001
  • Isolation and characterization of a novel IgD-binding protein from Moraxella catarrhalis
  • Lipopolysaccharide-binding protein increases toll-like receptor 4-dependent activation by nontypeable Haemophilus influenzae
  • Protein D expression promotes the adherence and internalization of non-typeable Haemophilus influenzae into human monocytic cells