Stefan Jovinge
Titel
Professor, Överläkare
Organisation
046-2220756
Stefan [dot] Jovinge [at] med [dot] lu [dot] se
Publikationer (hämtat ur Lunds universitets publikationsdatabas)
författare
- 2012
- An artificial neural network to safely reduce the number of ambulance ECGs transmitted for physician assessment in a system with prehospital detection of ST elevation myocardial infarction
- Cardiomyocyte Renewal in Humans
- Isolation of Cardiomyocyte Nuclei from Post-mortem Tissue.
- Myocardium at risk by magnetic resonance imaging: head-to-head comparison of T2-weighted imaging and contrast-enhanced steady-state free precession.
- 2011
- Myogenic Reprogramming of Bone Marrow Derived Cells in a WDmd Deficient Mouse Model.
- The evaluation of an electrocardiographic myocardial ischemia acuteness score to predict the amount of myocardial salvage achieved by early percutaneous coronary intervention Clinical validation with myocardial perfusion single photon emission computed tomography and cardiac magnetic resonance.
- 2010
- Brain natriuretic peptide is related to diastolic dysfunction whereas urinary albumin excretion rate is related to left ventricular mass in asymptomatic type 2 diabetes patients
- Cardiomyocyte cell cycle control and growth estimation in vivo- an analysis based on cardiomyocyte nuclei.
- Cardiovascular magnetic resonance of the myocardium at risk in acute reperfused myocardial infarction: comparison of T2-weighted imaging versus the circumferential endocardial extent of late gadolinium enhancement with transmural projection
- 2009
- Evidence for Cardiomyocyte Renewal in Humans
- Myocardium at risk after acute infarction in humans on cardiac magnetic resonance: quantitative assessment during follow-up and validation with single-photon emission computed tomography.
- Natriuretic peptides as indicators of cardiac remodeling in hypertensive patients.
- Tumor necrosis factor-alpha does not mediate diabetes-induced vascular inflammation in mice.
- 2008
- Cystatin C and cathepsins in cardiovascular disease.
- Elastin- and Collagen-Rich Human Carotid Plaques Have Increased Levels of the Cysteine Protease Inhibitor Cystatin C.
- Myeloid and lymphoid contribution to non-haematopoietic lineages through irradiation-induced heterotypic cell fusion.
- Suppression of stroke-induced progenitor proliferation in adult subventricular zone by tumor necrosis factor receptor 1.
- Turnover of Human Cardiomyocytes
- 2007
- Medial Expression of TNF-α and TNF Receptors Precedes the Development of Atherosclerotic Lesions in Apolipoprotein E/LDL Receptor Double Knockout Mice
- Medial expression of TNF alpha and TNF receptors preceds the development of atherosclerotic lesions in apolipoprotein E/LDL receptor double knockout mice
- 2006
- Engraftment of engineered ES cell-derived cardiomyocytes but not BM cells restores contractile function to the infarcted myocardium
- Glycaemic and nonglycaemic effects of pioglitazone in triple oral therapy of patients with type 2 diabetes.
- Glycemic and non-glycemic effects of pioglitazone in triple oral therapy in patients with type 2 diabetes mellitus
- 2005
- Absence of the protease inhibitor cystatin C in inflammatory cells results in larger plaque area in plaque regression of apoE-deficient mice.
- Conjugated linoleic acid increased C-reactive protein in human subjects
- Lack of the Cysteine-Protease Inhibitor Cystatin C Promotes Atherosclerosis in Apolipoprotein E-Deficient Mice.
- Nt-proANP in plasma, a marker of salt sensitivity, is reduced in type 2 diabetes patients.
- 2004
- Bone marrow-derived hematopoietic cells generate cardiomyocytes at a low frequency through cell fusion, but not transdifferentiation.
- Elevated Plasma Levels of Nt-proBNP in Patients With Type 2 Diabetes Without Overt Cardiovascular Disease.
- Inhibition of Tumor Necrosis Factor-{alpha} Reduces Atherosclerosis in Apolipoprotein E Knockout Mice.
- 2002
- Decreased inducibility of TNF expression in lipid-loaded macrophages.
- Increased expression of membrane type 3-matrix metalloproteinase in human atherosclerotic plaque: role of activated macrophages and inflammatory cytokines
- Inflammation and cholesterol
- Inhibitory effect on arterial injury-induced neointimal formation by adoptive B-cell transfer in Rag-1 knockout mice.
- Linoleic acid-stimulated vascular adhesion molecule-1 expression in endothelial cells depends on nuclear factor-kappaB activation.
- Supplementation with conjugated linoleic acid causes isomer-dependent oxidative stress and elevated C-reactive protein - A potential link to fatty acid-induced insulin resistance
- 1997
