Maria Björkqvist
Titel
bitr forskare
Organisation
046-222 05 25
Maria [dot] Bjorkqvist [at] med [dot] lu [dot] se
Publikationer (hämtat ur Lunds universitets publikationsdatabas)
författare
- 2013
- 2012
- Automated Behavioral Phenotyping Reveals Presymptomatic Alterations in a SCA3 Genetrap Mouse Model
- Bone Marrow Transplantation Confers Modest Benefits in Mouse Models of Huntington's Disease.
- Evaluation of a previously suggested plasma biomarker panel to identify Alzheimer's disease.
- Increased levels of hemoglobin and alpha1-microglobulin in Huntington's disease.
- No diagnostic value of plasma clusterin in Alzheimer's disease.
- 2011
- Abnormal peripheral chemokine profile in Huntington's disease.
- Gastrointestinal dysfunction contributes to weight loss in Huntington's disease mice.
- Hsa-miR-34b is a Plasma-Stable microRNA that is Elevated in Premanifest Huntington's Disease.
- Hsa-miR-34b is a plasma-stable microRNA that is elevated in pre-manifest Huntington's disease
- IGF-1 protects against diabetic features in an in vivo model of Huntington's disease.
- 2010
- 2009
- Beyond the brain: widespread pathology in Huntington's disease.
- Harnessing immune alterations in neurodegenerative diseases.
- Increased numbers of motor activity peaks during light cycle are associated with reductions in adrenergic alpha(2)-receptor levels in a transgenic Huntington's disease rat model
- Increased orexin levels in the cerebrospinal fluid the first year after a suicide attempt.
- Interleukin-6 Is Elevated in the Cerebrospinal Fluid of Suicide Attempters and Related to Symptom Severity.
- Mutant huntingtin interacts with {beta}-tubulin and disrupts vesicular transport and insulin secretion.
- 2008
- A novel pathogenic pathway of immune activation detectable before clinical onset in Huntington's disease
- A novel pathogenic pathway of immune activation detectable before clinical onset in Huntington's disease.
- Cocaine and amphetamine regulated transcript (CART) in suicide attempters.
- Increased metabolism in the R6/2 mouse model of Huntington's disease.
- Increased thirst and drinking in Huntington's disease and the R6/2 mouse
- Islet beta-cell area and hormone expression are unaltered in Huntington's disease.
- 2007
- Cocaine- and amphetamine-regulated transcript is increased in Huntington disease.
- Ghrelin and motilin are co-secreted from a prominent endocrine cell population in the small intestine.
- Ghrelin and motilin are cosecreted from a prominent endocrine cell population in the small intestine
- Orexin and psychiatric symptoms in suicide attempters.
- Reduced orexin levels in the cerebrospinal fluid of suicidal patients with major depressive disorder.
- Reduced orexin levels in the cerebrospinal fluid of suicidal patients with major depressive disorder.
- Testicular degeneration in Huntington disease
- 2006
- CART Regulates Islet Hormone Secretion and Is Expressed in the {beta}-Cells of Type 2 Diabetic Rats.
- Cerebrospinal fluid levels of orexin-A are not a clinically useful biomarker for Huntington disease.
- Hypothalamic-endocrine aspects in Huntington's disease.
- Progressive alterations in the hypothalamic-pituitary-adrenal axis in the R6/2 transgenic mouse model of Huntington's disease
- 2005
- Euploidy in somatic cells from R6/2 transgenic Huntington's disease mice
- Orexin loss in Huntington's disease.
- Reduction of GnRH and infertility in the R6/2 mouse model of Huntington's disease.
- Somatostatin, misoprostol and galanin inhibit gastrin- and PACAP-stimulated secretion of histamine and pancreastatin from ECL cells by blocking specific Ca(2+) channels.
- The R6/2 transgenic mouse model of Huntington's disease develops diabetes due to deficient {beta}-cell mass and exocytosis.
- 2003
- 2002
- 2001
- A-like cells in the rat stomach contain ghrelin and do not operate under gastrin control
- Cell-specific processing of chromogranin A in endocrine cells of the rat stomach
- Effects of CCK2 receptor blockade on growth parameters in gastrointestinal tract and pancreas in rats
- Gastrin and the neuropeptide PACAP evoke secretion from rat stomach histamine-containing (ECL) cells by stimulating influx of Ca2+ through different Ca2+ channels
