Madeleine Durbeej-Hjalt
Titel
professor
046-2220812
+46736407263
Madeleine [dot] Durbeej-Hjalt [at] med [dot] lu [dot] se
Publikationer (hämtat ur Lunds universitets publikationsdatabas)
författare
- 2012
- Cell-matrix interactions in muscle disease.
- Guidelines for the use and interpretation of assays for monitoring autophagy
- Increased Neointimal Thickening in Dystrophin-Deficient mdx Mice.
- Laminin-211 in skeletal muscle function.
- Porous protein-based scaffolds prepared through freezing as potential scaffolds for tissue engineering.
- 2011
- Autophagy is increased in laminin {alpha}2 chain-deficient muscle and its inhibition improves muscle morphology in a mouse model of MDC1A.
- Laminin isoforms in atherosclerotic arteries from mice and man.
- Proteasome Inhibition Improves the Muscle of Laminin {alpha}2 Chain Deficient Mice.
- Skeletal muscle laminin and MDC1A: pathogenesis and treatment strategies.
- Transgenic Expression of Laminin α1 Chain Does Not Prevent Muscle Disease in the mdx Mouse Model for Duchenne Muscular Dystrophy.
- 2010
- 2009
- E3 domain of laminin alpha 1 chain that binds to dystroglycan is not essential for survival and muscle regeneration in laminin alpha 1 chain mediated correction of laminin alpha 2 chain deficiency
- Intracellular signaling pathway alterations in laminin alpha 2 chain deficient skeletal muscle and brain
- Intrinsic laryngeal muscles are spared from degeneration in the dy(3k)/dy(3k) mouse model of congenital muscular dystrophy type 1A.
- Laminin alpha1 domains LG4-5 are essential for the complete differentiation of visceral endoderm.
- Substantial improvement of laminin alpha 2 chain deficiency by laminin alpha 1 chain overexpression persists throughout life
- 2008
- 2007
- Compositional differences between infant and adult human corneal basement membranes
- Dystroglycan: a possible mediator for reducing congenital muscular dystrophy?
- Gene expression profiling of differentiating embryonic stem cells expressing dominant negative fibroblast growth factor receptor 2.
- Laminin isoforms in development and disease.
- Role of the extracellular matrix and its receptors in smooth muscle cell function: implications in vascular development and disease
- 2006
- Dystroglycan is selectively cleaved at the parenchymal basement membrane at sites of leukocyte extravasation in experimental autoimmune encephalomyelitis.
- Extraocular muscle is spared upon complete laminin alpha2 chain deficiency: Comparative expression of laminin and integrin isoforms.
- Laminin alpha1 chain mediated reduction of laminin alpha2 chain deficient muscular dystrophy involves integrin alpha7beta1 and dystroglycan.
- Laminin {alpha}1 chain improves laminin {alpha}2 chain deficient peripheral neuropathy.
- Monoclonal anti-mouse laminin antibodies: AL-1 reacts with laminin alpha1 chain, AL-2 with laminin beta1 chain, and AL-4 with the coiled-coil domain of laminin beta1 chain.
- 2005
- 2004
- 2003
- 2002
