Webbläsaren som du använder stöds inte av denna webbplats. Alla versioner av Internet Explorer stöds inte längre, av oss eller Microsoft (läs mer här: * https://www.microsoft.com/en-us/microsoft-365/windows/end-of-ie-support).

Var god och använd en modern webbläsare för att ta del av denna webbplats, som t.ex. nyaste versioner av Edge, Chrome, Firefox eller Safari osv.

Explaining biological depression theories

Författare

Summary, in English

I AM GRATEFUL TO Dien Ho and James Phillips for their comments on my article (Mulinari, 2018). Although they approach the topic from different perspectives, they both seem to find my account of the evolution of monoamine theories into neuroplasticity theories to be compelling. They especially seem to find my principal argument to be persuasive: Until quite recently, the use of drugs to generate and test pathophysiological hypotheses—the pharmacological bridge—has been a paramount driving force in psychiatric research.

Avdelning/ar

Publiceringsår

2018-12-06

Språk

Engelska

Sidor

309-310

Publikation/Tidskrift/Serie

Philosophy, Psychiatry, and Psychology

Volym

4

Issue

25

Dokumenttyp

Artikel i tidskrift

Förlag

Johns Hopkins University Press

Ämne

  • History of Technology
  • Psychiatry
  • Sociology (excluding Social Work, Social Psychology and Social Anthropology)

Status

Published

Projekt

  • After the success with the new generation antidepressants: Experiences, practices, discourses and changes in the self.

ISBN/ISSN/Övrigt

  • ISSN: 1086-3303